Decision : Decision : The Committee considered this case for fixation of I O norms for the export product Aoprazolam under para 4.7 of HBP as per agenda.The Committee in light of the oral comments of the representative of PI Division in the meeting decided to fix I O Norms for the export product for the purpose of para 4.7 of HBP only as under: Export Import item 1. 2- Amino-5-Chloro Benzophinone --1.60 kg.
Profound oversedation Severe sedation due to the additive effect summation ; of drugs with sedating properties is a particular problem in elderly and frail people, and it can lead to falls and injuries especially fractures of the femoral neck ; . Excessively drowsy patients are also at increased risk of venous thromboembolism and, if confined to bed, of hypostatic pneumonia. In people who drive, increased sedation due to drug interactions carries a correspondingly increased risk of road traffic accidents. It is the responsibility of the individual not to drive if their ability to do so safely is impaired by drugs, whether prescribed or not. Driving when reactions are impaired by drugs may lead to prosecution. Profound and prolonged sedation can be brought about by inhibition of CYP3A4 enzymes that are involved in the metabolism of anxiolytics and sedatives. This occurs when one of the proteaseinhibiting antiviral compounds amprenavir, indinavir and ritonavir are co-administered with any of the following: alprazolam, clorazepate, diazepam, flurazepam and zolpidem. In each of these cases, the inhibition of metabolism causes high plasma levels of the anti-anxiety and hypnotic substances. Serotonin syndrome and related CNS toxicity Central nervous system toxicity refers to a wide range of drug-induced toxic effects, including excitation and restlessness; tremor, rigidity and myoclonus; pyrexia with sweating and flushing; fluctuating vital signs; and delirium. These can progress to stupor, coma and, at worst, death. The reactions include so-called serotonin syndrome, in which the toxicity is thought to be due to an increased effect of biogenic amines, particularly serotonin 5-hydroxytryptamine, 5-HT ; . Toxicity can also result from the combined use of drugs that increase monoamine neurotransmission. Examples are the toxicity resulting from the concomitant prescribing of monoamine oxidase inhibitors MAOIs ; with SSRIs, tricyclic and related antidepressants, tryptophan or St John's wort. It can also occur when any of these drugs is co-prescribed with.
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Mild antianxiety agents are occasionally used as an adjunct in treating chronic headaches to decrease muscle contraction or to treat anxiety symptoms during periods of extreme stress. They include alprazolam Xanax ; and clonazepam Klonopin ; . They tend to be highly addictive, however, and should therefore be used only on a short-term basis.
Table 3. Results of the sub-segmental Quantitative Coronary Angiographic QCA ; analysis n 96 ; In-stent Mean lumen diameter mm ; Before procedure After procedure At 6 months Minimal luminal diameter mm ; Before procedure After procedure At 6 months Stenosis % of luminal diameter ; Before procedure After procedure At 6 months Absolute gain Late loss mm ; 50% restenosis % of patients ; In- segment Proximal edge Distal edge.
We have also completed pilot pharmacokinetic studies for two antihistamine oral sprays loratadine and clemastine ; , an estradiol oral spray, an alprazolam oral spray and a progesterone oral spray. In addition, we completed phase 2 clinical trials for the clemastine oral spray. However, additional development work on these product candidates has been put on hold. We have also commenced formulation work on two new product candidates, tizanidine oral spray and ropinirole oral spray. Companies in the pharmaceutical and biotechnology industries have suffered significant setbacks in advanced clinical trials, even after obtaining promising results in earlier trials. Data obtained from tests are susceptible to varying interpretations which may delay, limit or prevent regulatory approval. In addition, companies may be unable to enroll patients quickly enough to meet expectations for completing clinical trials. The timing and completion of current and planned clinical trials of our product candidates depend on, among other factors, the rate at which patients are enrolled, which is a function of many factors, including: the number of clinical sites; the size of the patient population; the proximity of patients to the clinical sites; the eligibility criteria for the study; the existence of competing clinical trials; and the existence of alternative available products and altace.
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Patients may decide to forgo treatment for their illness. UNSUSPECTED TROUBLE WITH AN OTC MEDICATION A 58-year-old woman without a significant past medical history presented to her primary care physician with complaints of severe fatigue of 2 weeks' duration.9 She was an active woman who needed assistance with a wheelchair just the day prior to her examination. While the physical examination was negative except for scleral icterus, the laboratory evaluation revealed acute renal failure. The patient's serum creatinine was 3 mg dL 0.5 to1.4 mg dL ; when it had been at 0.9 mg dL just 2 months earlier. The hemoglobin and hematocrit were also low, possibly due to a total abdominal hysterectomy which required blood transfusion 3 months prior to this episode. Serum LDH was elevated as was the reticulocyte count. The urine was dark orange in color. When questioned about over-thecounter OTC ; medications, the patient revealed that she took acetaminophen and ibuprofen for migraine headaches. Her prescription medications included bupropion, alprazolam, levothyroxine, and omeprazole. The patient underwent a second interview to determine if there was anything else that she was taking OTC. At this point she remembered that she also took phenazopyridine on a regular basis, although no specific reason was given for her use of this agent. Phenazopyridine has been known to cause hemolytic anemia and renal failure. The drug was.
Similar tablets Error description. A patient noticed two different med- Conclusion recommendations ications in the prescription vial. The medications were Sometimes staffers are not aware that a potential probLipitor 10 mg for hypercholesterolemia ; and Zyrtec 10 lem exists or that an error has occurred. It's a good idea mg for allergic symptoms ; , both manufactured by Pfiz- to ensure that all staffers are notified of potential prober. The patient's prescription was for Zyrtec. A prescrip- lems by posting fliers and circulating newsletters such as tion for Lipitor was filled in the automatic counter first. the USP Quality Review that identify which products The counter may not have been fully emptied prior to may cause confusion. Sending e-mails, posting the inforfilling the Zyrtec prescription. The pharmacist attributed mation on your Intranet, or conducting an in-service the mix-up to the two products' similar tablet color and education program is also recommended. It is very size. Short staffing was also suggested as a contributing important to find ways to reach every person in your factor to this error. institution. Remember to include floating and temporary Reporting pharmacist's action recommendation. Check the staff, the night shift, and part-time employees. automatic counter for remaining tablets after each fill and THE AUTHOR is a clinical data analyst at USP Center for the Advancement of provide adequate staffing to enable double-checks to be Patient Safety. performed routinely. USP operates two complementary reporting programs: the Medication Error Reporting Program, presented in cooperation with the Error description. A patient was preInstitute for Safe Medication Practices, and MEDMARX. by scribed diazepam 2 mg, but received For more information on how to report errors, visit: by Susan Camp, Pharm.D. usp patientsafety. alprazolam 2 mg both are used to and ambien.
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Tions, benzodiazepines can be divided into two main groups on the basis of CYP 3A4 metabolism. The triazolobenzodiazepines, which include midazolam Versed ; , triazolam Halcion ; , and alprazolam Xanax ; , are dependent on CYP 3A4 for metabolism. Greenblatt et al.9 previously reported the effects of acute ritonavir Norvir ; exposure during alprazolam therapy. During the initial treatment with ritonavir, alprazolam clearance decreased, which led to enhanced alprazolam effects. Frye et al.10 studied ritonavir effects on alprazolam AUC during coadministration. After 12 days of ritonavir therapy, alprazolam AUC was diminished. This discrepancy is likely due to ritonavir's initial inhibition of CYP 3A4 followed by CYP 3A4 induction over time.11 Inhibiting CYP 3A4 will cause midazolam, triazolam, and alprazolam levels to rise, leading to possible toxicities such as oversedation, while induction of CYP 3A4 will cause therapeutic failure of the triazolobenzodiazepines, including withdrawal symptoms or dose escalation. Common CYP 3A4 inhibitors include the protease inhibitors; the nonnucleoside reverse transcriptase inhibitors delavirdine Rescriptor ; and efavirenz Sustiva and the psychotropics nefazodone Serzone ; , paroxetine Paxil ; , and fluoxetine Prozac ; . Common CYP 3A4 inducers include carbamazepine Tegretol ; , phenytoin Dilantin ; , rifampin Rifadin ; , and phenobarbital Phenob ; as well as nevirapine Viramune ; , a nonnucleoside reverse transcriptase inhibitor antiretroviral. Other benzodiazepines, such as lorazepam Ativan ; , oxazepam Serax ; , temazepam Restoril ; , and diazepam Valium ; , are metabolized by a variety of enzymes, including uridine 5 -diphosphate glucuronosyltransferase UGT ; 1A1, 1A3, and 2B7, with minor contributions by CYP 3A4. This multienzyme approach limits the frequency of drug-drug interactions. No case reports were found in the literature regarding interactions with antiretrovirals and these benzodiazepines. Flunitrazepam Rohypnol , "roofies" ; is a potent benzodiazepine not available in the United States but prescribed in Europe and Latin America. Common to the club scene, flunitrazepam can create a significant sense of euphoria and calm lending to its high abuse potential. Flunitrazepam is primarily metabolized by CYP 3A4 and 2C19. CYP 3A4 inhibitors such as ritonavir, paroxetine, and nefazodone will likely result in flunitrazepam toxicity, the effects of which include hypotension, confusion, visual disturbances, urinary retention, and aggressive behavior.12 Flunitrazepam is also a potent inhibitor of UGT 1A1, 1A3, and 2B7.13 This potent inhibition may result in increased.
Josefina g. Carbonell added "The financial independence and security of older people through the identification and prevention of Medicare fraud is vitally important. Senior Medicare Patrol volunteers across the country play an important role in educating our older Americans and their caregivers on how to identify and report suspected health care fraud." HHS has several programs to help Medicare beneficiaries protect themselves against fraud. The Senior Medicare Patrol Program, established by the Administration on Aging, educates and assists beneficiaries in protecting their Medicare information, detecting Medicare billing errors and reporting potential health care fraud and abuse. Instances of potential Medicare fraud also can be reported to the HHS Office of the Inspector general at1-800-HHS-TIPS 800-447-8477 ; or HHSTips oig.hhs.gov and amitriptyline.
Lorazepam brand name: ativan the most important information about lorazepam do not use this medication if you are allergic to lorazepam or to other benzodiazepines, such as alprazolam xanax ; , chlordiazepoxide librium ; , clorazepate tranxene ; , diazepam valium ; , or oxazepam serax.
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| Alprazolam clonazepamBenzodiazepines are central nervous system depressants that act in complex ways on the neurotransmitter GABA gamma-aminobutyric acid ; , which transmits messages from one brain cell to another. Directly or indirectly, benzodiazepines influence almost every brain function and ultimately most biological systems, including the central nervous, neuromuscular, endocrine and gastrointestinal. About 16 benzodiazepines are available in Canada today. Among the most common are Ativan lorazepam ; , Serax oxazepam ; , Rivotril and Klonopin clonazepam ; , Xanax alprazolam ; and Valium diazepam ; . Sleeping pills hypnotics ; such as Ambien zolpidem ; and Imovane zopiclone ; are not technically benzodiazepines, but they act by the same mechanisms and have the same effects on the brain and body. All benzodiazepines and sleeping pills are similar chemically but differ in potency equivalent doses may vary as much as twenty-fold ; , and the speed at which they are metabolized. These pharmacological differences affect symptoms and are important considerations in designing an appropriate tapering protocol for addicted individuals. Benzodiazepines have five short-term medical uses: anxiety relief anxiolytic ; , sleep hypnotic ; , muscle relaxant, anti-seizure anti-convulsant ; and pre-operative amnesiac ; . They are sometimes inappropriately prescribed for depression, but they are themselves central nervous system depressants and amoxicillin.
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1. 2. 7. Pharmacologic treatment of cocaine dependence Until now, none of the diverse psychopharmacologic treatments tested have demonstrated assured efficacy. It is notable that distinct pharmacological therapies have been developed on the basis of the modification of cerebral dopaminergic transmission. In general, two principal strategies have been studied. Dopamine receptor antagonist agents have been used to counteract cocaine's gratifying effects, and, in an opposite manner, agents that facilitate dopaminergic transmission have been used to counteract the dopamine deficit observed during withdrawal for a review, see Gorelick, 1995; McCance, 1997 ; . a. Dopaminergic antagonists Clinical studies that have evaluated the efficacy of dopaminergic receptor antagonists in the treatment of cocaine dependency are inconclusive, although some have indicated that dopaminergic receptor antagonists, primarily type D2, such as classical neuroleptics, can partially block the subjective effects of cocaine in humans and thereby reduce its consumption Berger et al., 1989; Sherer et al., 1989; Gawin et al., 1994; Khalsa et al., 1994 ; . Nevertheless, this therapeutic approach show two main problems. First, its chronic administration leads to the presence of anhedonia and undesireable extrapyramidal motor effects, resulting in an elevated level of treatment drop-out Kosten and Kleber, 1988; Decker and Ries, 1993 ; , and, second, repeated treatment with these pharmacologic agents can lead to an increase in dopaminergic postsynaptic receptor sensitivity, which, indirectly could increase the subjective effects of cocaine Goldfrank and Hoffman, 1991; Kosten, 1997 ; . Greater therapeutic potential is attributed to the antagonists D1, while with the antagonist D3 the opposite occurs. An 12, for example, buy cheap alprazolam.
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Abbott Laboratories have announced that it has received approval from the European Agency for the Evaluation of Medicinal Products EMEA ; to market its second-generation protease inhibitor PI ; , lopinavir ritonavir lopinavir r ; , previously known as ABT-378 r, for the treatment of HIVinfection. Lopinavir r is to marketed under the tradename Kaletra TM. The approval of lopinavir r was based on extensive clinical.
Benzodiazepines This drug class includes lorazepam, alprazolam, diazepam, clonazepam, temazepam, flurazepam, and others. A conscious person who presents to the ED with benzodiazepine intoxication usually is drowsy, unsteady, confused, disoriented, less alert, and impaired in judgment. In reality, the symptoms are not that different from those in patients with acute alcohol intoxication. When benzodiazepine intoxication is caused by a long-acting compound eg, clonazepam ; , the clinical signs of intoxication can continue for 24 hours. Abrupt discontinuation may result in tremors, anxiety, psychosis, confusion, and symptoms similar to those of delirium tremens. Discontinuation of short-acting benzodiazepines generates withdrawal symptoms within a few hours; sudden cessation of the long-acting compounds, however, may result in withdrawal effects that are delayed until the following day or even later.15, 16 Alcohol intoxication and withdrawal The effects of alcohol vary according to the level of consumption: low doses promote mild euphoria and uninhibited behavior, while substantial consumption may trigger irrational thinking, problematic behavior, psychomotor difficulties, and in rare cases, coma. The first step in evaluating a patient with a drinking problem is to identify possible predisposing factors for an episode of withdrawal.17, 18 These factors include high blood alcohol levels, history of withdrawal or seizure, concurrent use of sedating agents, and co-occurrence of acute or chronic medical problems. Most persons with alcohol dependence will experience mild signs and symptoms of withdrawal within 24 hours after the last drinking episode. These usually include restlessness, anxiety, tremors, tachycardia, GI discomfort, and insomnia. Symptoms may last for about a day and usually remit without major consequences. More rarely--but especially when predisposing factors are present--a mild withdrawal state may develop into 1 ; an episode of generalized seizure, typically within 24 to 72 hours or 2 ; delirium tremens, usually within 5 days of the last drink. Delirium tremens is characterized by a magnification of the symptoms of withdrawal and by the development of disorientation, visual hallu- cinations, high blood pressure, and in some cases, fever.17, 18 Stimulants: cocaine and amphetamines Initial signs of cocaine intoxication are restlessness, anxiety, hyperactivity, euphoria, dysphoria, elevated blood pressure levels, and increased heart rate. If the patient has taken high doses of cocaine, hallucinations and paranoid delusions may predominate the clinical picture. Overdose may lead to cardiac arrhythmias and extremely elevated blood pressure levels, which may be life-threatening. The euphoria related to cocaine use is sometimes followed by feelings of discomfort and depression and a craving that is usually defined as withdrawal.19, 20 Classified under "amphetamines" are stimulant drugs, such as methamphetamine and methylphenidate. Symptoms of acute amphetamine-related intoxication include decreased appetite, increased stamina and physical energy, irritability, aggressiveness, psychotic features, increased sexual drive, involuntary bodily movements, increased perspiration, hyperactivity, jitteriness, nausea, increased and irregular heart rate, and rarely, seizure. Withdrawal from amphetamines is usually characterized by depression, fatigue, withdrawn behavior, lack of motivation, and possibly, abdominal discomfort and headache, all of which can continue for several days.21, 22 Opioids The euphoric effects of opioids contribute to their widespread abuse. Heroin, morphine, codeine, oxycodone, and fentanyl are among the most commonly abused opioids. The most salient signs of intoxication include sedation, psychomotor difficulties, confusion, pinpoint pupils and, in more extreme cases, respiratory depression. If the opioid is discontinued, the withdrawal usually appears within a few hours for most of the drugs in this class and may last up to 1 week. The only exception is methadone, the withdrawal from which may appear the day after its discontinuation and tends to be milder and longer-lasting. Generally, the and aricept.
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Vascular endothelial growth factor VEGF ; is a hypoxia-inducible endothelial cell mitogen and survival factor. Its receptor VEGFR-2 KDR Flk-1 ; mediates these effects. We studied the expression of VEGF and VEGFR-2 in ischemic human and rabbit skeletal muscle by immunohistochemistry and in situ hybridization. Human samples were obtained from eight lower limb amputations because of acute or chronic critical ischemia. In chronically ischemic human skeletal muscle VEGF and VEGFR-2 expression was restricted to atrophic and regenerating skeletal myocytes, whereas in acutely ischemic limbs VEGF and VEGFR-2 were expressed diffusely in the affected muscle. Hypoxia-inducible factor-1 was associated with VEGF and VEGFR-2 expression both in acute and chronic ischemia but not in regeneration. Hindlimb ischemia was induced in 20 New Zealand White rabbits by excising the femoral artery. Magnetic resonance imaging and histological sections revealed extensive ischemic damage in the thigh and leg muscles of ischemic rabbit hindlimbs with VEGF expression similar to acute human lower limb ischemia. After 1 and 3 weeks of ischemia VEGF expression was restricted to regenerating myotubes and by 6 weeks regeneration and expression of VEGF was diminished. VEGFR-2 expression was co-localized with VEGF expression in regenerating myotubes. Macrophages and an increased number of capillaries were associated with areas of ischemic muscle expressing VEGF and VEGFR-2. In conclusion, two patterns of VEGF and VEGFR-2 expression in human and rabbit ischemic and atenolol and alprazolam, for example, akprazolam for dogs.
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The benzodiazepines BZD's, for short ; are a family of depressant drugs, discovered in 1957 and introduced throughout the world in the years since. Although the drugs first found fame for their "anxiolytic" anxiety-reducing ; effects, they're also prescribed as muscle relaxants, anticonvulsants, and sleeping pills. In general, though, BZD's are more alike than different. Main differences involve how fast they go to work, how long they remain active in the body, and how widely used they are. Like most depressants, BZD's are mainly prescribed to reduce anxiety and induce sleep. Unlike other drugs in the class, though, BZD's target receptors in the limbic region of the brain a key system in emotional control ; instead of depressing activity throughout the entire central nervous system. This means that BZD's produce their intended effects without most of the side effects--impaired thinking and judgment or serious respiratory depression--that are common among other depressants. Still, that doesn't mean they're harmless.
Of propoxyphene decreased the maximum plasma long term side effects from xanax of alprazolam by 6%, decreased clearance by 38%, long term side effects from xanax increased half-life by 58.
Where [open]Total is the total concentration of benzodiazepine which is in the ring-open form and [closed]Total is the total concentration of benzodiazepine which is in the ring-closed form at 37C Cyclodextrin pH Keq Aoprazolam No cyclodextrin 1 2 4 Midazolam 50 3 0.1 B0.1 100 6 0.1 Triazolam 20 2 B0.1 20 2 B0.1 50 3 B0.1 20 0.8 B0.1.
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Benzodiazepines those metabolized by cyp3a4, including alprazolam and triazolam ; : serum levels may be increased by troleandomycin; somnolence and confusion have been reported and altace.
THE ALTERNATIVES TO SMOKED MARIJUANA AS MEDICINE List compiled by Dr. Eric Voth, Fellow of the American College of Physicians ; Legalization advocates would have the public and policy makers incorrectly believe that crude marijuana is the only treatment alternative for masses of cancer sufferers who are going untreated for the nausea associated with chemotherapy, and for all those who suffer from glaucoma, multiple sclerosis, and other ailments. Numerous effective medications are, however, currently available for these conditions. There has been a recent study by the Institutes of Health to compare Metoclopramide with Marijuana to control vomiting and have found the former to 4 to times better than marijuana. Below is a list of the medications currently available for chemotherapy, and for all those who suffer from glaucoma, multiple sclerosis, and other ailments. Serotonin Antagonists Ondansetron Zofran ; Granisetron Kytril ; Tropisetron Navoban ; Dolasetron Phenothiazines Prochlorperazine Compazine ; Chlorpromaxine Thorazine ; Thiethylperazine Torecan ; Perphenazine Trilafon ; Promethazine Phenergan ; Corticosteroids Dexamethasone Decadron ; Methylprednisolone Medrol ; Anticholinergics Scopolamine Trans Derm Scop ; Butyrophenones Droperidol Inapsine ; Haloperidol Haldol ; Domperidone Motilium ; Benzodiazepines Lorazepam Ativan ; Qlprazolam Xanax ; Substituted Benzamides Metoclopramide Reglan ; Trimethobenzamide Tigan ; Alizapride Plitican ; Cisapride Propulsid ; Antihistamines Diphenhydramine Benedryl ; [SOURCE: 2001 WL 30659 Appellate Brief ; Brief of the Institute on Global Drug Policy of the Drug Free America Foundation; National Families in Action; Drug Watch International; Drug-free Kids: America's Challenge, et al., as Amici Curiae in Support of Petitioner Jan. 10, 2001, ; , U.S. v. Oakland Cannabis Buyers' Cooperative, 121 S.Ct. 1711 2001 ; and list reconfirmed on May 14, 2006]. This list was originally compiled by the Drug Free Schools Coalition and submitted to the Minnesota Legislature on February 14, 2007 by the Minnesota Family Council.
2-when to take h2ra regarding otc use for gerd only ; : patient can take 1 tablet when symptoms occur 2 ; , but when food is known to precipitate symptoms, h2ra should be taken an hour before food 1, 2.
Contraindicated for concurrent use decrease voriconazole levels ; : Rifampin, rifabutin, carbamazepine, phenobarbital. Voriconazole increases concurrent drug ; : Sirolimus, terfenadine, astemizole, cisapride, pimozide, quinidine, ergot derivatives Alter dose Cyclosporine: Increase cyclosporine, use half dose cyclosporine and monitor levels. Tacrolimus: Increase tacrolimus levels 2x; use 1 3 dose tacrolimus. Warfarin: Increase prothrombin time, monitor. Statins: Increase statin levels expected, reduce statin dose. Benzodiazepines: Midazolam, triazolam + alprazolam increased levels expected, reduce benzodiazepine dose. Calcium channel blocker: Felodipine level increase expected, may need dose decrease. Sulfonylureas: Tolbutamide, glipizide + glyburide level increases expected, monitor blood glucose. Vinca alkaloids: Vincristine + vinblastine levels increase expected, reduce dose to avoid neurotoxicity. Phenytoin: Decrease voriconazole, double oral dose of voriconazole and monitor phenytoin levels. Omeprazole: Levels double, reduce omeprazole to half dose.
Hydroxy alprazolam is formed in greater amounts than 4-hydroxy alprazolam in rat brain microsomal incubation, while this is not reflected in experiments using rat liver microsomes.
The dangerous performance enhancing steroids taken by athletes are male steroid hormonal drugs that build muscle. One of their risks is liver cancer. Similarly, female steroid hormonal drugs build breast tissue. They not only increase the risk of liver cancer but breast and cervical cancers as well. These powerful steroid drugs are taken by millions of teenage girls as BIRTH CONTROL PILLS. Why are teenage girls vulnerable to the cancer-causing effects of birth control pills?, because alprazolam dosages.
Korbsrisate S, Dumnin S, Chawengkirttikul R, Gherunpong V, Eampokalap B, Gongviseisoog C, Janyapoon K, Lertpocasombat K, Shimada T Distribution of Aeromonas hydrophila serogroups in different clinical samples and the development of polyclonal antibodies for rapid identification of the genus Aeromonas by direct agglutination. Microbiology and Immunology. 46 12 ; : 875-879, 2002. Aeromonas hydrophila, O-serogroup, agglutination test, polyclonal antibodies. We characterized a collection of 256 Aeromonas hydrophila strains isolated from blood, discharge and stool for their serogroup designation. Of these, 2.3% were untypable and 15.2% were rough strains. Among the typable strains, about 50% comprised serogroups O: 11, O: 16, O: 18, O: 34 and O: 83. To develop rapid differentiation of Aeromonas from other oxidase-positive bacteria, antisera against Aeromonas were produced to establish a direct, genus-specific, agglutination test. It was found that among 105 isolates of Aeromonas, 102 showed positive results with the agglutination test. The calculated sensitivity and specificity were 97.1% and 90.7%, respectively.
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